By B. Garreau
The 20th century used to be the century of the advance of morphological cerebral imaging by way of tomodensitometry (TDM) and Magnetic Resonance Imaging (MRI). in recent times new mind imaging tools have been utilized in adults with neurological lesions, and extra lately in adults with psychiatric problems. Now it's also attainable to take advantage of, almost all these morphological and useful mind imaging tools in children.
This publication provides the most morphological and practical mind imaging tools that we will use within the baby. major purposes are developped: physiopathological and therapeutical curiosity. The physiopathological strategy is of an outstanding curiosity, coupled with scientific overview in psychomotor problems like hyperkinetic or Tourette syndrom, and in developmental problems like autistic syndrom, psychological retardation, Rett Syndrom ...
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Extra resources for Neuroimaging in child neuropsychiatric disorders
Example text
After release into the synapse, acetylcholine is inactivated not by reuptake but by acetylcholine esterase enzymatic degradation. Glutamatergic system Glutamate and aspartate are excitatory amino acid neurotransmitters. They are involved in many mental and neurosensory functions, including cognition, memory, movement and sensation. They act on synaptic connection plasticity during development. In many diseases, excess glutamatergic system activation results in neuronal lesion and death (Lipton and Rosenberg, 1994).
Expansion Scientifique Francraise, Paris Ambrose J, Hounsfield G (1973) Computerized transverse axial tomography. British J Radiol 46: 148-149 Andreasen NC (1989) Nuclear magnetic resonance imaging. In: Andreasen NC (ed) Brain imaging: Applications in psychiatry. American Psychiatric Press, Washington DC, p 67-121 Angevine JB, Siman RL (1961) Autoradiographic study of cell migration during histogenesis of cerebral cortex of mouse. Nature 192: 766-773 Annett M (1980) Sex differences in laterality: meaningfulness versus reliability.
Citrulline + NO. Two series of experiments have shown the role of NO in perfusion-activity coupling. , 1989; Methods: principles, anatomical and physiological bases PET and SPECT Garthwaite, 1991}: this show that NO plays a role in the cellular response to activation of glutamate receptors. Secondly, the local increase in blood flow induced by NMDA is antagonised by NO synthase inhibitors (Faraci and Breese, 1993). NO might therefore mediate adaptation of perfusion during neuronal activity, as shown in figure 3.