By David H. Durrant

Richly illustrated and simple to take advantage of, Myelopathy, Radiculopathy, and Peripheral Entrapment Syndromes offers wide and updated information regarding the etiopathic and scientific presentation of neurovascular compression insult to the spinal twine, spinal nerve roots, and peripheral nerves. Written with either the scholar and practitioner in brain, the e-book covers myelopathy, radiculopathy, and peripheral entrapment syndromes in separate sections, making the data effortless to discover.

Each part includes:

  • Introduction
  • Relevant nearby anatomy
  • Pathophysiology and mechanism of the injury
  • Clinical indicators and symptoms
  • Electrodiagnostic features
  • Diagnostic imaging
  • Syndromes particular to the extent of compromise

    More than 275 designated, unique illustrations and MRIs let you visualize the recommendations. greater than one hundred thirty concise tables disguise the topic-specific highlights. those gains mix to offer the booklet a quick-reference caliber that units it aside. Myelopathy, Radiculopathy, and Peripheral Entrapment Syndromes presents all of the functional info you would like - in a single handy source - to successfully diagnose and think again neurological problems of the backbone and extremities from a neurologic perspective.

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    Extra info for Myelopathy, Radiculopathy, and Peripheral Entrapment Syndromes

    Sample text

    11 There is little doubt in the scientific community over the production of free radicals and lipid peroxidation following SCI; however, elucidation of the complex interactions and exact biochemicals involved in the process is far from complete. 1 Selective Pathophysiologic Mechanisms in SCI • • • • • • • • • • Free radical-mediated cell injury Cation-mediated cell injury Excitatory amino acids (glutamatergic toxicity) Intracellular-mediated apoptosis Aberrant calcium fluxes Lipid peroxidation Accumulation of arachidonic acid metabolites (eicosanoids) Ischemia/infarction Edema Endogenous opiate receptor activation receptors.

    First-order axons enter the spinal cord along the dorsolateral tract of Lissauer (lateral root entry zone) at the posterior horn and synapse with second-order neurons. Second-order neurons lie within the deeper layers of the posterior horn and give rise to axons that decussate in the ventral white commissure just anterior to the central spinal canal, converging within the lateral funiculus to form the spinothalamic tract. Secondorder neurons ascend contralaterally within the anterolateral funiculus to synapse within the VPL nucleus of the thalamus and within the posterior interlaminar nuclei of the thalamus.

    M. J. W. ) Handbook of Clinical Neurology, Vol. XII. Vascular Diseases of the Nervous System. Amsterdam: North Holland, 478–491, 1972. 29. A. C. Ischemic myelopathy: a review of spinal vasculature and related clinical syndromes. Arch. Phys. Med. , Apr. 73:365–372, 1992. 30. , The cytoarchitectonic organization of the spinal cord in the cat, J. Comp. , 96:415–495, 1952. 31. , Disorders of the spinal cord and cauda equina, Curr. Opin. Neurol. , 6(4):545–548, 1993. 32. , The Human Nervous System: Structure and Function, Philadelphia, PA: Williams & Wilkins, 1996.

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    Myelopathy, Radiculopathy, and Peripheral Entrapment by David H. Durrant
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