By M.A. Sahraian, Ernst-Wilhelm Radü, A. Gass, S. Haller, L. Kappos, J. Kesselring, J.-I. Kira, K. Weier
MRI has turn into the most paraclinical attempt within the analysis and administration of a number of sclerosis. now we have proven greater than four hundred photographs of other general and abnormal MS lesions during this atlas. each one photo has a educating element. New diagnostic standards and differential analysis were mentioned and the booklet is supported by means of a instructing DVD for you to see MS lesions in several slices and sequences.
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Extra resources for MRI atlas of MS Lesions
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5. 6. Gawne-Cain ML, O'Riordan JI, Coles A et al (1998) MRI lesion volume measurement in multiple sclerosis and its correlation with disability: a comparison of fast fluid attenuated inversion recovery (fFLAIR) and spin echo sequences. J Neurol Neurosurg Psychiatry 64:197–203 Hajnal JV, De Coene B, Lewis PD et al (1992) High signal regions in normal white matter shown by heavily T2-weighted CSF nulled IR sequences. J Comput Assist Tomogr 16:506–513 Rydberg JN, Hammond CA, Grimm RC et al (1994) Initial clinical experience in MR imaging of the brain with a fast fluid-attenuated inversion-recovery pulse sequence.
AJNR Am J Neuroradiol 27:1165–1176 8. Goodin DS (2006) Magnetic resonance imaging as a surrogate outcome measure of disability in multiple sclerosis: have we been overly harsh in our assessment? Ann Neurol 59:597–605 9. Kidd D, Barkhof F, McConnell R et al (1999) Cortical lesions in multiple sclerosis. Brain 122:17–26 10. O’Riordan JI, Thompson AJ, Kingsley DP et al (1998) The prognostic value of brain MRI 34 1 MS Lesions in T2-Weighted Images 11. 12. 13. 14. 15. in clinically isolated syndromes of the CNS: a 10-year follow-up.
2001). Ring-like enhancement probably arises from recent inflammation at the periphery of an ac- tive lesion in which the blood-brain barrier defect has been partially or completely repaired in the center (Bastianello et al. 1990). It is also noted that ongoing activity affects one margin of the plaque, and the remainder is quiescent, which may be a cause of the formation of arc pattern (He et al. 2001). Nodular enhancing lesions tend to differ in size and also to decrease their size over time.