By Randell C. Alexander, James W. Hanson (auth.), Louise R. Greenswag R.N., PH.D., Randell C. Alexander M.D., PH.D. (eds.)
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Additional resources for Management of Prader-Willi Syndrome: Under the Sponsorship of The Prader-Willi Syndrome Association
Example text
J. (1981). Neurology of the newborn. B. Saunders. , & Hajra, A. (1986). Zellweger syndrome: Diagnostic assays, syndrome delineation and potential therapy. American Journal of Medical Genetics, 21,69-82. Zellweger, H. (1983). The floppy infant: A practical approach. Helvetica Paediatrica Acta, 38,301-306. Zt1llweger, H. (1987). Peroxisomal disorders. Developmental Medicine and Child Neurology, 29,821-829. , & Ionasescu, V. (1973). Early onset of myotonic dystrophy. American Journal of Diseases of Children, 125, 601-605.
Medicine, 62, 59-80. , & Mukaida, C. (1985). Osteoporosis in Prader-Willi syndrome. American Journal of Human Genetics, 37, A49. , & Sitruk-Ware, R. (1986). Biological effects of estradiol-17f3 in postmenopausal women: Oral versus percutaneous administration. Journal of Clinical Endocrinology and Metabolism, 62, 536-54l. , & Kauli R. (1982). Primary gonadal failure and precocious adrenarche in a boy with Prader-Labhart-Willi syndrome. European Journal Pediatrics, 139, 201-203. B. (1973). Testosterone treatment for micropenis during early childhood.
Peroxisomal disorders. Developmental Medicine and Child Neurology, 29,821-829. , & Ionasescu, V. (1973). Early onset of myotonic dystrophy. American Journal of Diseases of Children, 125, 601-605. 3 Hypogonadism and Osteoporosis KAREN RUBIN and SUZANNE B. CASSIDY Hypogonadism has been a recognized major feature of Prader-Willi syndrome (PWS) since it was first described (Prader, Labhart, & Willi, 1956). 1). Osteoporosis is a recently described finding in PWS that is likely to be an additional consequence of hypogonadism, at least in part.