By Firas H. Kobeissy
Every 12 months, an predicted 1.7 million american citizens maintain mind damage. long term disabilities effect approximately half average mind harm survivors and approximately 50,000 of those situations bring about death.
Over the decade, the sphere of neurotrauma has witnessed major advances, specifically on the molecular, mobile, and behavioral degrees. This growth is basically a result of advent of novel ideas, in addition to the advance of recent animal versions of principal worried procedure (CNS) damage.
Brain Neurotrauma: Molecular, Neuropsychological, and Rehabilitation Aspects presents a accomplished and updated account at the most modern advancements within the zone of neurotrauma, together with mind damage pathophysiology, biomarker study, experimental versions of CNS harm, diagnostic tools, and neurotherapeutic interventions in addition to neurorehabilitation suggestions within the box of neurotraum examine.
The booklet contains a number of sections on neurotrauma mechanisms, biomarker discovery, neurocognitive/neurobehavioral deficits, and neurorehabilitation and remedy ways. moreover, a bit is dedicated to types of light CNS harm, together with blast and sport-related injuries.
This ebook, with its diversified coherent content material, is a invaluable and accomplished reference for people drawn to the various and heterogeneous points of CNS pathology and/or rehabilitation needs.
Read or Download Brain Neurotrauma: Molecular, Neuropsychological, and Rehabilitation Aspects PDF
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Extra resources for Brain Neurotrauma: Molecular, Neuropsychological, and Rehabilitation Aspects
Example text
Increased attention to TBI related to surveillance and clinical follow-up by the VA and the DOD; these joint initiatives aimed to detect and treat TBI/concussion to prevent and possibly to mitigate their persistent effects (Management of Concussion/mTBI Working Group, 2009). PTSD, depression, pain, and substance abuse accompanying mTBI pose challenges for management and disability assessment. , 2014; Taylor, 2011). Among these estimates of prevalence and coprevalences of combat-associated conditions, about 6% of these veterans had all three diagnoses.
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27:1059–1068. P. Becker, T. A. Hovda. 1990. Massive increases in extracellular potassium and the indiscriminate release of glutamate following concussive brain injury. Journal of Neurosurgery. 73:889–900. , W. E. Thomas. 2004. Traumatic brain injury in the United States: Emergency department visits, hopitalizations, and deaths. Centers for Disease Control and Prevention, National Center for Injury Prevention and Control, Atlanta, GA. C. L. K. McIntosh. 2001. The duality of the inflammatory response to traumatic brain injury.
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