By P T K Woo; J F Leatherland
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1990) and tattered, sloughing epithelium (Schaperclaus, 1951) are seen in some cases. Rhizocysts anchor the trophont to its host and damage the epithelium. This damage may be exacerbated by the hydrodynamic forces exerted on the trophont projecting into the water moving past the fish. Cellular damage is thought to attract parasites because trophonts typically cluster in damaged areas. Gill histopathology ranges from separation of the respiratory epithelium to severe hyperplasia of the entire filament.
Whether tricaine has the same effect on oodinid dinoflagellates is unknown. , 2001). , 1998a, b). Sequencing of the SSU-rRNA genes from three geographical isolates of A. , in preparation). J. G. Levy a PCR assay. , Solana Beach, California, USA) followed by PCR. The assay was also specific for Amyloodinium, potentially allowing for highly sensitive monitoring of pathogen load in susceptible populations. Prevention and Control Amyloodinium The economic importance of warmwater mariculture has created an effort towards the development of methods for the prophylaxis and treatment of amyloodiniosis.
Are located in the mucus or on/in epithelial cells and extracellular fluid of the epithelium. Serum from blue tilapia (Oreochromis aureus) with no previous exposure to Amyloodinium has anti-Amyloodinium activity in a cell culture infectivity assay. , 1992). Heating the serum to 47°C for 20 min or 56°C for 30 min, as well as treating it with carageenan or zymosan, suggested that complement-like activity was responsible for at least part of the activity. Amyloodinium is also highly sensitive to natural, host-produced antibiotics, known as histone-like proteins (HLPs).