By Ze'ev Hochberg
Working with ideas from the fields of evolutionary and developmental biology (evo-devo), this attention-grabbing paintings bargains a brand new method of reading baby progress and improvement, interpreting every one level and transition intimately, from fetal improvement to preadulthood. in response to the author's in-depth assessment of the present literature and his personal observations as a pediatric endocrinologist, the publication demonstrates how the transitions among human lifestyles heritage levels signify special sessions of evolutionary adaptive reaction to the surroundings. moreover, the writer explains why an figuring out of those transition classes permits us to raised comprehend the series and mechanisms of kid progress in addition to to higher diagnose baby progress disorders.
Logically geared up and obviously written, Evo-Devo of kid Growth:
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Sets an exceptional origin of rules akin to evolutionary pondering in drugs and baby progress, lifestyles background concept, and heterochrony and allometry
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Examines the connection among baby progress and the speculation of lifestyles history
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Applies evo-devo idea to fetal development, infancy, adolescence, juvenility, early life, and preadulthood
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Explores the trade-offs and adaptive phenotypic plasticity in the course of transition periods
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Explains the position of lifestyles background concept in realizing and diagnosing development problems comparable to Down syndrome, Noonan syndrome, and Silver-Russell syndrome
In addition to the author's personal research and observations, this booklet additionally gains notes from prime clinicians and evolutionary biologists, delivering extra views at the dating among evo-devo and baby progress and development.
Evo-Devo of kid progress offers a brand new viewpoint for evolutionary biologists to appreciate the stages and transitions of kid progress. in addition, it deals a brand new method of support clinicians to raised comprehend and diagnose a extensive variety of kid development disorders.
Content:
Chapter 1 creation (pages 1–10):
Chapter 2 baby progress and the idea of existence heritage (pages 11–52):
Chapter three Fetal progress (pages 53–66):
Chapter four Infancy (pages 67–90):
Chapter five adolescence (pages 91–95):
Chapter 6 Juvenility (pages 96–114):
Chapter 7 early life (pages 115–126):
Chapter eight formative years (pages 127–130):
Chapter nine Evolutionary thoughts for physique dimension (pages 131–142):
Chapter 10 strength concerns (pages 143–148):
Chapter eleven level Transitions: Trade?Offs and Adaptive Phenotypic Plasticity (pages 149–178):
Chapter 12 lifestyles historical past conception in realizing development issues (pages 179–186):
Chapter thirteen whilst the applications fall apart (pages 187–189):
Chapter 14 Concluding comments (pages 190–193):
Read Online or Download Evo-Devo of Child Growth: Treatise on Child Growth and Human Evolution PDF
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Additional info for Evo-Devo of Child Growth: Treatise on Child Growth and Human Evolution
Example text
And osteopenia/osteoporosis. CRH = corticotropin-releasing hormone; AVP = arginine-vasopressin; ACTH = corticotropin; F = glucocorticoid cortisol; NE = norepinephrine; E = epinephrine; iCRH = immune CRH; IL-6 = interleukin-6; GH = growth hormone; IGF-1 = insulin-like growth factor 1; LH = luteinizing hormone; T = testosterone; FSH = follicle-stimulating hormone; E2 = estradiol; P4 = progesterone; TSH = thyrotropin; T3 = triodothyronine; TG = triglycerides; LDL = low density lipoprotein cholesterol; HDL = high density lipoprotein cholesterol; APR = acute phase reactants such as C-reactive protein; ABP = arterial blood pressure.
This is an innate program of the organism that normally unfolds during a systemic inflammatory reaction, and includes somnolence, fatigue, nausea, and depressive affect, and occurs concurrently with activation of the acute phase reaction by the liver and stimulation of the sensory afferent nervous system manifest as hyperalgesia and fatigue. The peripheral secretion of “immune” CRH by postganglionic sympathetic nervous system neurons and the norepinephrine-activated release of IL-6 by peripheral immune and other cells, respectively lead to degranulation of mast cells in 36 CHILD GROWTH AND THE THEORY OF LIFE HISTORY several tissues (Chrousos 1995, 2000).
Atypical depression, respectively, are both associated with development of the metabolic syndrome X and its cardiovascular and other sequelae through two opposed but metabolically converging pathways (Chrousos 2007; Gold, Gabry et al. 2002) (Fig. 7A). In melancholic depression-like states, we have the standard consequences of chronic stress hormone hypersecretion and, in parallel, chronic insulin hypersecretion, resulting in visceral fat accumulation and sarcopenia, as outlined above. , de facto insulin hypersecretion, a state that also favors visceral fat accumulation, with all that this entails.